Finally, to wrap up our genetic analyses, let’s take a look at our last norn to import and a new baby I’m introducing.
Hoss – Male – Son of Cassy and David
Moniker – 1NAB
Look at Handsome Hoss! I do love seeing Horse Norns cropping up, even if only on the Creature’s face. I just love those teeny, tiny little horns that the sport with their stylish haircuts. He had a neat mix of parts, too.
When I investigated his genetics, I did find one interesting gene that had popped up in the past. At 1 hour old, he wasn’t even old enough to breed, but I was excited for his offspring already.
Good | Bad | Interesting
229 Different in Hoss 38 0 Emb B MutDupCut 2*<219> + 4*Glucose => 1*Antibody3 + 1*Hotness; half-life = 80.
229 Different in dad4 38 0 Emb B MutDupCut 2*Antigen3 + 4*Glucose => 1*Antibody3 + 1*Hotness; half-life = 80.
After mulling over this genetic mutation, I came to realize just how much of a blessing in disguise it was. If you think back to my post about Typhoid Mallory, you might remember that she had a mutation that prevented her body from ‘reacting’ to Antigen 2. Basically, she wasn’t able to have an immune response til she was at life-stage 16, which doesn’t exist and isn’t auto-corrected by the game engine. While this means she can harbor disease, it means that she is also safe from it. Isn’t that handy?
Hoss on the other hand has the same mutation as his father. Instead of a non-existent life stage protecting him from Antigen 3, however, he reacts to chemical 219, which isn’t injected in the game. Just as good as being immune, the way I see it.
Archie – Male – Son of Cassy and David
Moniker – 7GXR
Next was Archie, who I named during his vocabulary lessons. He’s Hoss’s full brother, so I’m happy to see more Horse faces in the game. He’s a cheerful little guy, too and I’m happy to welcome him to the group. Peony came and distracted him for a while, but we eventually got through lessons and he got to taste cheese and carrots before very long.
Good | Bad | Interesting
229 Different in Hoss 38 0 Emb B MutDupCut 2*<219> + 4*Glucose => 1*Antibody3 + 1*Hotness; half-life = 80.
229 Different in dad4 38 0 Emb B MutDupCut 2*Antigen3 + 4*Glucose => 1*Antibody3 + 1*Hotness; half-life = 80.
What luck! Archie had the same mutation as his brother Hoss. This gave me some hope to breed a line of norns that were immune to this antigen. Every little bit helps, you know?
With all the investigating out of the way, I was finally able to continue documenting their lives, just as before. It wasn’t that I couldn’t blog without looking at their genetics, but after looking at Mallory’s genes, I guess curiosity got the better of me.
I, too, love those Horse Norn faces! Which also reminds me that I really need to finish up my spriting project… They’re prone to odd pink face shadows, and some weird white pixels in their hair. But I digress!
I actually thought that this type of mutation is highly dangerous, although I could be completely wrong! C1 will randomly infect a Norn with a certain mix of antigen(s) and toxins. Feel free to correct me, but wouldn’t this mean that antigen 3 would keep them sick forever? It wouldn’t be a problem if they were only infected with antigen 3, but any histamines or toxins would be stuck in their system. It seems like they attach to an antigen and only go away when the antigen is fought off. Would this mutation make it impossible for them to produce any antibody 3 to get rid of an antigen 3 illness?
I’m a complete nerd when it comes to C1 illnesses, but please feel free to point at me, laugh, and tell me I’m completely wrong. Ha! It’s happened many times before. In any event, I find this mutation very interesting. More importantly, I’m looking forward to seeing how things play out for Hoss and Archie!
Really? The mutation does make it impossible for them to produce antibodies to fight the illness, but at the same time, it removes the hotness symptom. I’ll need to spend more time with this gene to really understand how it works (trust me, just assume I’m a complete newbie at all this – because I am), but I’m glad you’ve got me thinking about it!
I’m thinking of getting more involved in studying C1 illnesses, because I still don’t have a complete grasp on them, either! I think that this mutation will help in removing the hotness symptom, but any other toxins that might attach to the antigen would (theoretically, at least to me) be stuck in the Norn’s system. So you could have persistent coughing/sneezing, or perpetual fever/sleep toxin. I think I might start a more in-depth study on these illnesses… I think you and I had talked about some sort of collaborative project, so perhaps this could be part of that! I’m sure we’ll talk more soon. And if I’m completely wrong in what I’m thinking here, please let me know! C1 illnesses are simple in some ways, but can get really complicated when you factor in various genetic mutations.